Allergic Diseases by Patterson

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J Allergy Clin Immunol 1998;101:683–690. MacDonald SM, Lichtenstein LM, Proud D, et al. Studies of IgE dependent histamine releasing factors: heterogeneity of IgE. J Immunol 1987;139:506. Siriganian RP. Mechanism of IgE-mediated hypersensitivity. In: Middleton E, Reed CE, Ellis EF, et al, eds. Allergy: principles and practice. 4th ed. St. Louis: CV Mosby, 1993. Dvorak AM, Morgan ES. Ribonuclease-gold ultrastructural localization of heparin in isolated human lung mast cells stimulated to undergo anaphylactic degranulation and recovery in vitro.

Basophils are also a prominent source of IL-4 and IL-13 ( 56). The preponderance of these mast cell and basophil cytokines can be categorized as proinflammatory (IL-1, IL-6, TNF-a); possess properties important in IgE synthesis (IL-4, IL-13); stimulate eosinophil growth, longevity, localization, and activation (IL-3, IL-5, and GM-CSF); and participate in airway remodeling (IL-9). MEDIATOR INTERACTIONS The mediators generated and released after mast cell activation have been isolated, identified, and characterized as individual factors, whereas physiologic and pathologic events reflect their combined interactions.

6. 7. 8. 9. 10. 11. 12. 13. 14. 15. 16. 17. 18. 19. 20. 21. 22. 23. Williams CMM, Galli, SJ. The diverse potential effector and immunoregulatory roles of mast cells in allergic disease. J Allergy Clin Immunol 2000;105:847–859. Bingham CO, Austen KF. Mast-cell responses in the development of asthma. J Allergy Clin Immunol 2000;105:S527–S534. Bernstein JA, Lawrence ID. The mast cell: a comprehensive, updated review. Allergy Proc 1990;11:209. Irani AA, Schechter NM, Craig SS, et al. Two types of human mast cells that have distinct neutral protease compositions.

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